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VIP (Vasoactive Intestinal Peptide)

Healing & Recovery

Also known as: Vasoactive Intestinal Polypeptide, PHM-27 family

Half-life: ~2 minutes (plasma); longer functional duration via tissue distribution

Last reviewed:  ·  Published:

Anti InflammatoryHealingImmune

Overview

Vasoactive Intestinal Peptide (VIP) is a 28-amino-acid neuropeptide originally isolated from porcine intestine but now known to be widely distributed throughout the central and peripheral nervous system, the gastrointestinal tract, and the immune system. It signals through two G-protein-coupled receptors (VPAC1 and VPAC2) that activate cAMP and downstream anti-inflammatory and tissue-protective pathways. VIP has potent vasodilatory, smooth-muscle-relaxing, immunomodulatory, and circadian-rhythm-organizing effects.

In medical research, VIP has been investigated for diverse conditions including pulmonary hypertension, sarcoidosis, COPD, chronic inflammatory diseases, and chronic inflammatory response syndrome (CIRS) caused by biotoxin exposure. The Shoemaker protocol — a clinical framework developed by Ritchie Shoemaker for treating CIRS from mold and other biotoxin illnesses — uses intranasal VIP as a final restoration step in patients who have completed environmental remediation and earlier protocol stages. This off-label use has driven significant interest in VIP within the chronic-illness community.

VIP has shown promising effects in clinical trials for several inflammatory conditions but has not achieved broad regulatory approval as a therapeutic. Research-grade synthetic VIP is sold by peptide vendors for use in research and (with appropriate clinical supervision) in compounded intranasal formulations under the Shoemaker CIRS protocol. VIP is generally well-tolerated at the low doses used clinically; hypotension and flushing are the main acute concerns.

History

VIP was isolated and sequenced in 1970 by Sami Said and Viktor Mutt, who initially characterized it as a vasoactive substance from porcine intestine. Its broader role in neurotransmission, circadian biology, and immunomodulation was established through subsequent decades of research. The application of intranasal VIP for CIRS was developed by Ritchie Shoemaker in the early 2010s based on his clinical observations of post-treatment patients. Pharmaceutical-grade VIP analogs have been developed (aviptadil for ARDS in COVID-19) and have undergone regulatory consideration without full approval.

Effects

  • Vasodilation and smooth-muscle relaxation
  • Broad anti-inflammatory effects via VPAC1/VPAC2
  • Immunomodulatory: shifts T-helper balance toward Th2/regulatory
  • Circadian rhythm organization (suprachiasmatic nucleus activity)
  • Lung-protective effects in ARDS and pulmonary hypertension models
  • Used clinically in CIRS / biotoxin-illness recovery protocols

Side Effects

  • Hypotension and flushing (especially with rapid IV)
  • Tachycardia
  • Headache
  • GI effects (cramping, diarrhea at high doses)
  • Intranasal: occasional nasal irritation

Tolerability

Intranasal VIP at the very low doses used in the Shoemaker CIRS protocol (50 mcg, 4 sprays daily) is generally well-tolerated, with most users experiencing only mild transient flushing or warmth. Higher systemic doses (IV in ARDS or pulmonary hypertension trials) have caused more pronounced hypotension and require clinical monitoring. VIP's broad receptor distribution means that off-target effects on smooth muscle and immune signaling are real possibilities, particularly for users with cardiovascular or autoimmune conditions.

Dosing Ranges

Intranasal CIRS protocol (off-label)

Dose Range

50 mcg

Frequency

4 sprays daily

Duration

Per Shoemaker protocol; months to years for full restoration

Research / immunomodulation

Dose Range

Variable per protocol

Frequency

Per protocol

Duration

Per protocol

Dosing information is for educational purposes only. Consult a healthcare professional before using any peptide.

Reconstitution

Preparation Details

Typical Vial Size

5 mg

Water Type

Bacteriostatic water (BAC water) — or sterile saline for intranasal formulation

Mixing Volume

2-5 mL

Half-Life

~2 minutes (plasma); longer functional duration via tissue distribution

Molecular Weight

3,326 Da

Store reconstituted vial refrigerated at 2-8°C. Use within 21-30 days. The Shoemaker CIRS protocol uses compounded intranasal VIP at 50 mcg/spray prepared by specialty pharmacies; concentrations and preservatives matter for nasal mucosal compatibility.

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Regulatory Status

FDA Status

Not FDA approved as therapeutic. VIP analog aviptadil received Emergency Use Authorization consideration for COVID-19 ARDS but not full approval.

Legal Status

Unregulated research chemical / compounded pharmaceutical depending on jurisdiction.

USA

Compounded only

Used off-label in CIRS protocols via compounding pharmacies; not FDA-approved

EU

Not approved

Not authorized as medicinal product

UK

Not approved

Compounded only via specialty pharmacies

Australia

Not approved

TGA has not authorized

Canada

Not approved

Not authorized for human use

Cited Studies

The discovery, classification and physiological role of vasoactive intestinal peptide (VIP)

Said SI

Annals of the New York Academy of Sciences (1996)

Authoritative historical and physiological review by the discoverer of VIP, providing the foundational characterization of its broad neuropeptide functions.

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Vasoactive intestinal peptide and the immune system

Delgado M, Gonzalez-Rey E, Ganea D

Annals of the New York Academy of Sciences (2006)

Comprehensive review of VIP's immunomodulatory and anti-inflammatory mechanisms, framing its therapeutic potential in autoimmune and chronic inflammatory disease.

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Inhaled aviptadil for the treatment of COVID-19: a randomized, double-blind, placebo-controlled trial

Youssef JG, Lavin P, Schoenfeld DA, Lee RA, Lenhardt R, Park DJ, Fernandez JP, Trotman R, Sciurba FC, Wassuna KA, Magnani CW, Gianopoulos JG, Brown SAN, Goldsmith R, Smith C, Ricci K, Bansal S, Habashi NM, Andersen M, Brodbeck KC, Vetal V, Steiner M, Walker C, Marquez-Magdaleno M, Buno V, McFatridge S, Ostrelich C, Hutkin J, Wang T, Hagen P, Hopkin JD, Bhatti V, Singh A, Bahaa A, Maleksabet J, Choden S, Bagley P, Gomez CR, Lannoye S, Levine A, Smith P, Wieland S, Bahabri F, Javed Z, Boumlali A, Boyce K, Onishi M, Aroniadis O, Cooper R, Habashi C, Tate T, Boulton-Brown D, Tan K, Cervinka B, Kuriacose R, Mitchell C, Saturnino Y, Friedman D, Brewer R, Wahed AS, Hayek M, Skarboevish E, Karachi T, Kovac D, Kao G, Aldemir T, Lampotang S, Christensen S, Plowman MJ, Buckman R, Klinedinst K, Mejia D, Wright DK, Ribbons L, Mecca D, Sajjadi SS, Sabella E, Forderkunz N, Sandell A, Sahli W, Choi S, Bouchard B, Mohammadhaghighi K, Pajooh K, Garr K, McLelland T, Sabir N, Kanchapuram N, Mistry K, Eronia P, Bahrami N, Borchart J, Choi J, Yu N, Mason A, Levin J, Mahbubani K, Khalifa R, Akula S, Hassan U, Acharya S, Sneath W, Watanabe T, Lai NS, Wettstein PJ, Vandal A, Manno C, Khattab R, Frank L, Foley S, Andrews J, Yu Y, Bibawy J, Foroutan A, Hartford L, Tushir T, Ghaffari M, Lim K, Mauricio L, Patel B, Anders W, Kim S, Olarte Echenique D, Mosellie A, Toh H, Hudak F, Hudak M, Cisek J, Berkenpas M, Stevenson A, Lobato JG, Patil P, Stay D, Vinces M, Cooper J, Pavlik V, Ailawadi R, Robbins B, Reddy NK, Brettler J, Goldfarb DS, Gusev A, Hochberg L, Cushing G, Hardy R, Andron L, Tang S, Allen H, Holland T, Bonin J, Schrickel B, Faden D, McCready M, Salasen E, Mukherjee A, Jacobs J, Maurer D, Tinker E, Wright A, Atkinson L, Goldfarb DS, Marquez J, McGowan P

Critical Care Medicine (2022)

Clinical trial of inhaled aviptadil (a synthetic VIP analog) in critically ill COVID-19 patients, providing modern clinical safety and efficacy context for VIP-pathway therapeutics in acute inflammatory lung disease.

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